Black Cumin Provides New Definitions for Physiology and Pathology
| dc.contributor.author | Mosab Nouraldein Mohammed Hamad | |
| dc.date.accessioned | 2023-11-21T08:59:26Z | |
| dc.date.available | 2023-11-21T08:59:26Z | |
| dc.date.issued | 2022 | |
| dc.description.abstract | Human Immunodeficiency Virus infects a type of white blood cell in the body’s immune system called a T-helper cell (also called a CD4 cell). These vital cells keep us healthy by fighting off infections and diseases. It cannot reproduce on its own. Instead, the virus attaches itself to a T-helper cell and fuses with it (joins together). It then takes control of the cell’s DNA, makes copies of itself inside the cell, and finally releases more HIV into the blood. HIV will continue to multiply and spread throughout the body – a process called the HIV lifecycle. In this way, HIV weakens the body’s natural defences and over time severely damages the immune system [1]. Viral proteins, termed "virotoxins," are released from the infected glial cells that initiate a cascade of positive feedback loops by activating uninfected microglial cells and astrocytes. These activated cells release a variety of toxic substances that result in neuronal dysfunction and cell loss | |
| dc.identifier.uri | https://ds.eaeu.edu.sd/handle/10.58971/387 | |
| dc.language.iso | other | |
| dc.publisher | جامعة الشيخ عبدالله البدري | |
| dc.title | Black Cumin Provides New Definitions for Physiology and Pathology |